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They certainly can't reverse if the genetic material integrates into the genome. However certain symptoms can at least be treated.

Dr Cory , Dr. Marick et al have had success with using Ivermectin for post vaccine injury

https://covid19criticalcare.com/wp-content/uploads/2022/11/I-RECOVER-Post-Vaccine-Treatment-Summary.pdf

As with any protocol these compounds dosage and what other compounds to take with it are important, there are no single magic bullet mono-therapies.

For example Ivermectin because of its antis-spike protein properties combined with doxycycline has evidence of being efficacious with prion based diseases. Doxycycline has excellent fibril disaggregation properties as well as down regulation of hyperphosphorylation of the Tau protein.

Doxycycline has evidence of being efficacious with prion-based diseases. Doxycycline has excellent fibril disaggregation properties as well as downregulation of hyperphosphorylation of the Tau protein.

Medina L, González-Lizárraga F, Dominguez-Meijide A,

et al. Doxycycline interferes with tau amyloid aggregation

abolishing its associated neuronal toxicity. bioRxiv. 2020.

Doxycycline interferes with tau amyloid aggregation abolishing its associated neuronal toxicity

Doxycycline interferes with tau amyloid aggregation abolishing its assoc...

Tauopathies are neurodegenerative disorders with increasing incidence and still without cure. The extensive time...

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fighting β-amyloid oligomers and neuroinflammation.

Frontiers in Pharmacology. 2019; 10: 738.

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6769662/

Dominguez-Meijide A, Parrales V, Vasili E, et al. Doxycycline

inhibits α-synuclein-associated pathologies in vitro and in

vivo. Neurobiology of Disease. 2021; 151: 105256.

Doxycycline inhibits α-synuclein-associated pathologies in vitro and in vivo - PubMed

Doxycycline inhibits α-synuclein-associated pathologies in vitro and in ...

Parkinson's disease (PD) and dementia with Lewy bodies (DLB) are neurodegenerative disorders characterized by th...

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doxycycline for treating synucleinopathies Evidence from a

pre-clinical mouse model. Parkinsonism Related Disorders.

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Repositioning doxycycline for treating synucleinopathies: Evidence from a pre-clinical mouse model - PubMed

Repositioning doxycycline for treating synucleinopathies: Evidence from ...

Our findings promote Doxy as a valuable multi-target therapeutic approach counteracting both symptoms and neurop...

Paldino E, Balducci C, La Vitola P, et al. Neuroprotective

effects of doxycycline in the R6/2 mouse model of Huntingtons

disease. Molecular neurobiology. 2020; 57: 1889-1903.

Neuroprotective Effects of Doxycycline in the R6/2 Mouse Model of Huntington’s Disease

Neuroprotective Effects of Doxycycline in the R6/2 Mouse Model of Huntin...

Mechanisms of tissue damage in Huntington’s disease involve excitotoxicity, mitochondrial damage, and inflammati...

Lavigna G, Masone A, Bouybayoune I, et al. Doxycycline

rescues recognition memory and circadian motor rhythmicity

but does not prevent terminal disease in fatal familial insomnia

mice. Neurobiology of Disease. 2021; 158: 105455.

https://pubmed.ncbi.nlm.nih.gov/34358614/

Varges D, Manthey H, Heinemann U, et al. Doxycycline

in early CJD a double-blinded randomised phase II and

observational study. Journal of Neurology Neurosurgery

Psychiatry. 2017; 88: 119-125.

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Doxycycline in early CJD: a double-blinded randomised phase II and obser...

EudraCT 2006-003934-14; Results.

. Lu Y, Yang Y, Chen W, et al. Minocycline but not doxycycline

attenuates NMDA-induced Ca2+i and excitotoxicity.

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Minocycline, but not doxycycline attenuates NMDA-induced [Ca2+]i and excitotoxicity - PubMed

Minocycline, but not doxycycline attenuates NMDA-induced [Ca2+]i and exc...

Minocycline and doxycycline, two semisynthetic second-generation tetracyclines, are reported to provide neuropro...

. Noble W, Garwood CJ, Hanger DP. Minocycline

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2712603/

Medina L, González-Lizárraga F, Dominguez-Meijide A,

et al. Doxycycline interferes with tau amyloid aggregation

abolishing its associated neuronal toxicity. bioRxiv. 2020.

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